1. Conclusion Troglitazone induced mixed type of cell death in HeLa cells.
结论上述结果表明,曲格列酮可以引起混合型细胞死亡。

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2. The flavonoids extracts had strong growth inhibitory effects on Hela cells.
菜芙蓉总黄酮具有抑制肿瘤细胞生长的作用。

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3. Partial HeLa cells presented characteristic morphological changes of apoptosis.
光镜下部分细胞呈现典型凋亡形态学改变。

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4. The expression in HeLa cells could induce the cell apoptosis instead of senescence.
钙调素基因在真核细胞中表达没有引起肿瘤细胞的凋亡和衰老。

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5. The expression of cloned gene in HeLa cells did not induce the cell apoptosis or senescence.
钙调素基因在真核细胞中表达没有引起肿瘤细胞的凋亡和衰老。

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6. The FCM analysis showed that the apoptosis rate increased after Hela cells were effected by matrine.
流式细胞仪检测到细胞在苦参碱作用后,细胞凋亡率增加;

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7. Flow cytometry revealed that E1A transfection induced s stage suppression and G2 stage arrest in Hela cells.
流式细胞术结果显示:转染e1 A基因后,细胞周期出现S期抑制,G 2期阻滞。

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8. Through RT-PCR analysis of the expression about the HDM2 protein in Hela cells, a new HDM2 splice variant was identified.
对克隆到的HDM2基因片段进行测序分析,从中筛选到一种新的HDM2剪接变异体。

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9. When the genetic bio-computer was introduced into the different cell types, only HeLa cells, but not the healthy ones, were destroyed.
当基因生物微机被导入这两种不同的细胞类型时,只有海拉细胞被摧毁,而正常细胞则安然无恙。

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10. Thenthe activity of HeLa cells was tested by MTT method and the cells were stained by acridine orange (AO) and observed by microscopy.
经相应的温度和时间处理后,以四甲基噻唑兰(MTT)比色法检测细胞活性,吖啶橙荧光染色法观察细胞的形态学改变。

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11. The protective effects of N acetyl L cysteine (NAC) and glutathione (GSH) were studied in the HeLa cells exposed to sulfur mustard (HD).
本文在细胞水平上研究了N-乙酰L-半胱氨酸(NAC)及还原型谷胱甘肽(GSH)对抗硫芥致细胞坏死及凋亡的效应。

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12. Hela cells presented the characteristic morphological changes of apoptosis under inverted microscope and transmission electron microscope.
透射电镜观察发现部分细胞发生凋亡早期改变。

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13. The mechanism of DAPT inhibited the proliferation and prompted the apoptosis of SiHa and HeLa cells may be due to decreased the formation of NICD.
DAPT能抑制宫颈癌细胞的增殖,促进宫颈癌细胞的凋亡,其机制可能与抑制NICD的生成有关。

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14. The recombinant strain also had lower adhesiveness(P>0.05)and significantly lower relative invasiveness to the HeLa cells than its wild type strain(P
细胞试验表明重组菌对细胞的黏附力和相对侵袭力均有不同程度的降低,而相对侵袭力与野生型菌株具有显著性差异(P

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15. The effects of AFP could be blocked by anti AFP monoclonal antibody. The results indicated that AFP could regulate the growth of HeLa cells through enhancing the expression of these oncogenes.
AFP的作用均可被抗AFP单克隆抗体所拮抗 。实验结果提示 ,AFP对细胞生长的调节作用可能通过促进这些原癌基因的表达来实现 。

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16. Their effects on growth inhibition against HL-60, HeLa and SGC-7901 cells were determined by SRB assay.
用SRB法测定目标化合物对癌细胞的增殖抑制作用。

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17. Their effects on growth inhibition against HL-60, HeLa and SGC-7901 cells were determined by SRB assay.
用SRB法测定目标化合物对癌细胞的增殖抑制作用。

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